Click on the link below to access a variety of forms
Horse Health Free Reports
Click on the link below to access a variety of forms
Horse Health Free Reports
Due to Draft horses being prone to certain diseases such as, metabolic ailments like PSSM (Polysaccharide storage myopathy), laminitis, Cushings, founder, tying-up, and shivers diet is imperative. Based on these ailments, starch and sugar calories should be replaced by fiber and fat calories.
It is recommended that these guys have high quality forage and some concentrates while working due to their slower metabolism (similar to ponies). Meaning that the less energy they use, the more weight they gain. High carbohydrate feed should be avoided, as a forage with a rational balancer and/or a low NSC feed.
Calorie Breakdown:
Calculating Fat Content:
Pounds of feed per day x % of fat
For example,
3 lbs of feed= 3 x 0.25= 0.75 lb fat
Feed Brands:
Feeds should have no more than 33% sugar and starch (low carb).
Low in starch and sugars: soy, beat pulp, wheat bran, wheat middlings
Feeds with 20%+ of fat should be supplemented with rice bran (20% fat). Feed with anything less than 20% should be supplemented with 100% additional fat source.
Supplements:
The Plan
Mix 12 parts alfalfa (or Purina or Southern States Feed or mix of the two) with 1 part water. Soak for 10 minutes. Add in oil. Let it sit for 2+ hours. Right before feeding add in the supplement (Vet E/Selenium).
While agreeing to save the ponies from slaughter, I saw a beautiful, senior mare. She is a 17 hand, 17-year old, Belgian mare who was an Amish workhorse until put into the auction. She is visibility underweight and her coat is dull and patchy but her eyes bright and soulful. I watched to see if she got rescued as her slaughter date was for 5/7/2020. Well, last night, I took the plunge and purchased the sweet girl.
Ever since losing Chance I have not had a desire to get another horse. Over a year ago a neighbor gave me a thoroughbred mare but shortly after she arrived I realized I was not ready and I had just learned I was pregnant. Fortunately, I was able to find her a wonderful forever home. However, upon coming across this Belgian I knew she was my next heart horse. The thing is I only have one paddocked fenced in that holds three miniature donkeys. So I am quickly getting things in order- scheduled to have another paddock fenced in, a large run-in dropped off, and everyones vaccinations up-to-date. One of my neighbors has a barn and paddocks and no other horses on the property and generously offered to allow my new mare and friend’s two ponies quarantine there for 30 days. By the time quarantine is over, the fence and run-in will be up and ready for the new members of the family!
There is also a ton of research that I need to do about owning a draft horse. They are a special breed and more susceptible to metabolic disorders and a ton of hoof issues. Once again I will be using this platform as a way to organize my ongoing research. Wish me luck!
The other day I was outside with a girlfriend and her two boys (6 ft apart) as they were feeding one of my miniature donkeys, Trou, a carrot. They turned and said, “That one is missing a tooth!” I smiled and upon realizing that they said went over to look for myself. Sure enough my Trou has a cracked, half missing tooth! I did a quick check- no cuts, swelling, abrasions, no puffiness, heat, and he did not seem to be in any pain. I called the vet and explained what was going on and that I needed them to come out to pull my donkey’s tooth since it was cracked so close to the gum line (like a human would have done). They came out two days later and simply said “it will grow back.” I was completely shocked! “It will what?!” The vet explained that equine (horse, mule, donkey) teeth grow. They have a very long root that as the tooth wears down, it continues to grow. I asked why a horse that cribs ends up having nubs for teeth. The vet explained that due to cribbing a horse will use up their “reserve” faster than most other horses so by the time they hit their late 20’s they no longer have any growth left. Sure enough, a week later, I checked on Trou’s tooth and it was almost back to normal!
For more information on equine teeth click on the link below:
https://www.thesprucepets.com/learn-about-your-horses-teeth-1885784
Pivoting During a Pandemic
— Read on horsenetwork.com/2020/04/pivoting-during-a-pandemic/
Seven states remain affected by vesicular stomatitis virus.
— Read on thehorse.com/178754/usda-officials-confirm-63-newly-affected-vesicular-stomatitis-premises/
Learn equine biosecurity basics for the farm, horse show, and breeding shed to protect your horses from infectious diseases.
— Read on thehorse.com/features/practical-biosecurity-tips-to-protect-your-horse/
#1 Abdominal Pain, Colic Signs Perform Whole Horse Exam™ (WHE) Assess Color of Mucous Membranes Assess Demeanor or Attitude Assess Gut or Intestinal Sounds Assess Manure Assess Capillary Refill Time (CRT) by examining Gums Give Intramuscular (IM) Injection Give Oral Medication Sand Sediment Test…
— Read on horsesidevetguide.com/Common+Horse+Emergencies+and+the+Skills+You+Need+to+Help
With freezing temperatures comes the need for extra care and attention for horses and other equids.

The growing season some parts of the nation had last year produced overly stemmy or fibrous hay with a lower digestibility. As a result, making certain that horses are supplemented with grain when fed lower quality hay will help them maintain body weight and condition, a key factor in withstanding cold temperatures.
Constant access to clean, fresh water at 35 to 50°F is an absolute necessity to keeping horses healthy. This can be achieved via heated tanks or buckets, or by filling a tank, letting it freeze, cutting an access hole in the frozen surface, and then always filling the tank to below the level of the hole from that point on. This provides a self-insulating function and will typically keep the water below from freezing. Regardless of the method you choose, it’s important to check tanks frequently to ensure your horse’s water remains free of ice.
Additional ways to keep horses comfortable in cold weather include making sure they have access to shelter. A well-bedded, three-sided shed facing south or east will typically provide adequate protection from wind and snow, as can appropriate bluffs or treed areas.
When the temperatures get colder, mature horses will not typically move around much in an effort to conserve energy. Making an attempt to keep hay, shelter, and water fairly close together can limit the energy expenditure required, thus conserving body condition.
And, finally, keeping horses at a body condition score of 5 or 6 (on a 9-point scale) will help prevent surprises when horses shed their winter hair in the spring, and improve conception rates for those choosing to breed.
By Dr Sarah-Jane Wilson, Livestock Biosecurity Network Northern Regional Manager
Death, unfortunately, is one of the most inescapable elements of life and one that, when it occurs within the livestock production chain, raises a litany of biosecurity issues.
Animal carcasses can pose a serious risk to both human and animal health, can jeopardise biosecurity and impose a range of environmental impacts if not properly disposed of. These risks can include polluting water courses, spreading disease and interfering with community amenity.
The old practice of simply leaving a carcass anywhere in the paddock to rot simply does not stack up in a modern livestock industry where the implications of incorrect carcass management are better understood.
In fact, depending on where you live, there may be local, state or national regulatory requirements that relate to your on-farm waste management procedures and I encourage you to make yourself familiar with these obligations.
We strongly recommend carcass disposal is integrated into your on-farm biosecurity plan and that you consider the methods available to dispose of animal carcasses or animal waste products including hide, gut or bones after home slaughter or wool that is not suitable for baling. Another important consideration is the equipment you may need to assist in this disposal.
If you live on a small farm, your best alternative may be to engage a specialist disposal service as opposed to burial or on-site burning. Again, there may be some regulatory requirements for producers in higher density areas and I encourage you to seek the advice of your local council or departmental staff to ensure you adhere to any applicable guidelines. Generally speaking burial is often the most practical and preferred method of disposal on a small farm if you do not have access to a disposal service.
For all producers, your geographic location and common endemic diseases should be taken into consideration. For example, if you live in a botulism affected area, burning is the recommended and preferred method. Botulism spores can live in the soil for many years, so simply burying the carcass will not suffice.
If you have multiple sudden deaths in your herd or flock, and/or do not know the cause of death, then it is best practice to investigate. Your local veterinarian or animal health/biosecurity officer may be able to provide further information. If you suspect an emergency or unusual disease, you should report this as soon as possible to your local animal health authority.
For more information, the NSW Environmental Protection Agency and the Tasmanian Environmental Protection Agency provide some good advice, as do most of the other applicable state departments, on how to effectively and responsibly dispose of the livestock carcasses on your property.
Top tips
Choosing a site (Source: NSW EPA)
If the carcasses must be disposed of on-site, it is preferable to have:
Other pit considerations (Source: Tas EPA)
If you need to burn (Source: NSW EPA)
Planning ahead for what to do with a carcass or, multiple carcasses in the event of a natural disaster, can substantially reduce the stress of the moment. It can also make a dramatic contribution to the biosecurity soundness of your property and our greater livestock industries.
Here at LBN we’ve designed a small template to assist producers in thinking through the options that best work for them. This can be found at: http://www.lbn.org.au/farm-biosecurity-tools/on-farm-biosecurity-planning-tools/.
Ends
Written by Dr. Tom Lenz on behalf of AQHA
Few horse owners are aware of this disease which is a progressive, paralyzing disease that is 80-100% fatal in affected horses. Botulism is sometimes referred to as “forage poisoning” in adult horses or “shaker foal syndrome” in foals. The disease is caused by a potent toxin that is produced by the bacterium Clostridium botulinum. This bacterium lives in the soil as well as the intestinal tract of many normal birds and mammals, including the horse. It produces dormant spores that can be found in 18.5% of soil samples tested in the United States. The disease is most prevalent in Kentucky, Ohio, Maryland, Pennsylvania, California, and Tennessee although it can occur in any state in the U.S. Clostridium botulinum produces several different toxins. Type A toxins are often implicated in human infant botulism and are most often found west of the Rocky Mountains. Types B, C, and D toxins are usually involved in cases of equine botulism, with Type B responsible for 85% of horse cases in the U.S.
Horses of any age are susceptible to botulism which may be initiated by one of three ways. In the case of “forage poisoning” the horse ingests toxins that are contaminating feedstuffs such as grain or hay. Feed contamination is most often due to putrefied carcasses of birds or rodents. A Type C botulism outbreak that killed a number of horses in California several years ago was traced back to hay that contained the infected carcass of a rabbit. The bacteria can also enter a horse’s body via contamination of a wound, especially a deep puncture wound. A good example is “Shaker foal syndrome” which is most frequently caused by the bacteria entering the newborn foal’s body through the foal’s moist navel. Something that can be minimized by dipping the foal’s navel in mild iodine solution soon after birth. The third method in which the disease can be initiated is by ingestion of the spores in the soil. The ingested spores activate in the horse’s intestinal tract where they produce potent toxins that are then absorbed. Regardless of the route, once the bacteria have entered the horse’s body they produce toxins that block transmission of nerve impulses to the horse’s muscles. This results in a progressive paralysis of all the major muscle groups and is concluded with paralysis of the diaphragm, which results in death. Once symptoms develop, death may ensue in several hours or take up to a week.
The disease is difficult to diagnose because it resembles several other medical conditions and diseases such as choke, colic, rabies, EPM, and sleeping sickness. Blood samples very rarely contain toxin and necropsy following the death of the horse usually does not provide a conclusive diagnosis. Because the bacteria often occur naturally in the horse’s intestinal tract, isolation of the organism from the sick horse’s intestine is not diagnostic.
Clinical signs of the disease in adult horses suffering from “forage poisoning” initially include loss of facial expression, a sleepy appearance, saliva drooling from the corner of the mouth, loss of tongue control and loss of tail tone. The horse’s appetite is good, but it has a great deal of difficulty in chewing food and appears to be “playing” in their feed and water buckets. As muscular weakness becomes more profound, the horse will experience muscle trembling, generalized sweating and labored breathing. A weakened, shuffling gait may develop and the horse may take stiff, short steps as if walking on eggs. Eventually, the horse goes down and death results due to paralysis of the respiratory muscles. “Shaker foal syndrome” is usually seen in foals one to two months of age but can develop as early as two weeks or as late as 8 months of age. Early signs in foals are similar to those seen in adults in that the foal shows generalized weakness, poor tail tone, and loss of tongue control. The foal will often dribble milk from the mouth and nostrils because of an inability to swallow. Because of muscle weakness, the foal will lie down frequently. When it does rise, it soon develops muscle tremors and collapses. Affected foals may die within 12 hours of exhibiting symptoms or may linger for as long as a week.
Botulism is usually fatal if left untreated. Prior to the advent of antitoxin, the death rate among affected foals was greater than 90%. With the use of antitoxins in conjunction with antibiotics and supportive therapy, the mortality rate can be reduced to less than 25%. Animals unable to swallow should be fed through a nasogastric tube and placed on IV fluids. Once the toxin produced by the bacteria is attached to the nerve ending it cannot be neutralized by the antitoxin. Therefore, early treatment is critical. Even with aggressive therapy, recovery is slow and may require up to two weeks before the affected horse recovers.
Because of the high death rate and the difficulty in diagnosing this disease, prevention through vaccination is critical. A Type-B Toxoid vaccine is available and is quite effective in preventing the disease. In areas where the disease is prevalent, pregnant mares should be initially vaccinated at the 8th, 9th and 10th month of gestation and thereafter at the 10th month of each pregnancy. Yearly vaccination of adults in areas where the disease frequently occurs is also recommended. If unable to vaccinate the mare prior to foaling, limited information suggests that foals vaccinated with the toxoid at 2 weeks, 4 weeks and at 8 weeks of age developed adequate protection, even in the presence of passive maternal antibodies. Currently, no licensed vaccines are available for preventing botulism due to Cl. botulinum types A or C or other subtypes of toxins. Cross-protection between subtypes does not occur.
As in all horse health issues, your local veterinarian is your best source of information.
ABOUT THE AUTHOR: Thomas R. Lenz, DVM, M.S., Diplomate of the American College of Theriogenologists, is a trustee of the American Horse Council, past chairman of AQHA’s research committee and past president of the American Association of Equine Practitioners. This article is provided courtesy of AAEP Alliance Partner, AQHA.
Reviewed and updated by original author in 2016.